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G. MARK BAILLIE, PHARM.D., MHA, is Clinical Coordinator, Surgery and Critical Care Pharmacy Services, and Clinical Associate Professor of Pharmacy, Medical University of South Carolina, 150 Ashley Avenue, Charleston, SC 29425 bailliem musc ; . Based on the proceedings of a symposium held December 9, 2003, during the 38th ASHP Midyear Clinical Meeting, New Orleans, LA, and supported by an unrestricted educational grant from Roche Laboratories, Inc. Dr. Baillie received an honorarium for his participa.

23. AHFS Drug Information - 2007; p. 688 24. AHFS Drug Information - 2007; pp. 688-9 25. AHFS Drug Information - 2007; p. 689 26. AHFS Drug Information - 2007; p. 689 27. USP DI - 2005; p. 1256 28. AHFS Drug Information - 2007; p. 690 29. AHFS Drug Information - 2007; p. 691 30. USP DI - 2005; p. 1257 31. AHFS Drug Information - 2007; pp. 691-2 32. AHFS Drug Information - 2007; p. 692 33. AHFS Drug Information - 2007; p. 690 34. USP DI - 2005; p. 1257 35. AIDSinfo - Important New Clinical Data, Potential Early Virologic Failure Associated With the Combination Antiretroviral Regimen of Tenofovir Disoproxil Fumarate, Didanosine, and Either Efavirenz or Nevirapine in HIV Treatment-Naive Patients with High Baseline Viral Loads. [Dear Healthcare Provider Letter]. New York: Bristol-Myers Squibb; November 2004. Available at: : aidsinfo.nih.gov other DHCP11Nov04 . Accessed 06 30 07. Bristol-Myers Squibb - Sustiva Prescribing Information, January 2007, pp. 13-4. Available at: : bms products data index . Accessed 06 30 07. USP DI - 2005; p. 1257 38. USP DI - 2005; p. 1259 39. Bristol-Myers Squibb - Sustiva Prescribing Information, January 2007, p. 36. Available at: : bms products data index . Accessed 06 30 07. Bristol-Myers Squibb - Sustiva Prescribing Information, January 2007, p. 45. Available at: : bms products data index . Accessed 06 30 07. ChemIDplus - Available at: : chem.sis.nlm.nih.gov chemidplus chemidlite . Accessed 06 30 07. ChemIDplus - Available at: : chem.sis.nlm.nih.gov chemidplus chemidlite . Accessed 06 30 07. ChemIDplus - Available at: : chem.sis.nlm.nih.gov chemidplus chemidlite . Accessed 06 30 07. Merck Index - 2006; p. 598 45. Bristol-Myers Squibb - Sustiva Prescribing Information, January 2007. Available at: : bms products data index . Accessed 06 30 07. Bristol-Myers Squibb - Sustiva Prescribing Information, January 2007, p. 2. Available at: : bms products data index . Accessed 06 30 07. Bristol-Myers Squibb - Sustiva Prescribing Information, January 2007, p. 2. Available at: : bms products data index . Accessed 06 30 07. ChemIDplus - Available at: : chem.sis.nlm.nih.gov chemidplus chemidlite . Accessed 06 30 07. ChemIDplus - Available at: : chem.sis.nlm.nih.gov chemidplus chemidlite . Accessed 06 30 07. ChemIDplus - Available at: : chem.sis.nlm.nih.gov chemidplus chemidlite . Accessed 06 30 07. ChemIDplus - Available at: : chem.sis.nlm.nih.gov chemidplus chemidlite . Accessed 06 30 07.

Defendants. The specific allegations and the referenced Exhibits state the Plaintiffs' factual basis for claiming that the specific Defendants have knowingly reported or caused the reporting of false price representations to Medi-Cal. Specifically, the Plaintiffs have listed each drug at issue, specific industry insider price information available to Ven-A-Care, specific price information obtained in the Attorney General's investigation and the false prices that the specific Defendants knowingly caused to be reported. A comparison of the prices generally and currently available to industry insiders such as Ven-A-Care, with the prices reported to MediCal, reveals that the reported prices were false and misleading. The Medi-Cal reimbursement system for the Defendants' drugs was based upon the reported prices and each Defendant knew this, yet caused the reporting of the false and misleading prices that they knew would be used by Medi-Cal to determine reimbursement amounts. A. 50. SPECIFIC ALLEGATIONS AS TO DEFENDANT ABBOTT From on or after January 1, 1994, to the present, Defendant ABBOTT knowingly!

Allegations 4. i ; D and 4. ii ; G relate to the provision of drugs unnecessarily and or without taking reasonable steps to ensure there was a therapeutic need for them. Professor Mullen gave evidence that although he questioned, for example, antiretroviral therapy. For oral dosage form tablets ; : for high blood pressure: adults— at first, 10 milligrams mg ; or 20 mg once a day.

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Investigators at UCSF are using array CGH to study many types of solid tumors and leukemias. The goals of the research include finding the locations of new genes that are important in the development and progression of cancer and determining how specific genetic abnormalities affect patient outcome and response to therapy. One benefit of studying DNA is that it remains fairly stable even in tumor tissue preserved for years. Measurement of DNA from these preserved specimens is particularly informative since the clinical history of these tumors already is known. Research investigations aim not only to find individual genetic abnormalities, but also to identify patterns exhibited by various tumor types. Such investigations should lead to the identification of the basic biological mechanisms that have failed and allowed multiple aberrations to occur in tumors. Ultimately, it is hoped, it will be possible to more successfully target these mechanisms to improve treatment. Researchers at UCSF and elsewhere also are using "expression microarray" studies, based on the measurement of another genetic molecule, messenger RNA, to gauge how active genes are in tumor cells versus normal cells and digoxin, for instance, saquinavir. Submitted by law enforcement agencies. These units in labs across the state serviced 14, 429 cases during 2003. Evidence handled included firearms, drugs, biological DNA and toxicology specimens, trace items, latent fingerprints, items associated with computer crimes and questioned documents.
Abacavir sulfate ABC ; Ziagen, GlaxoSmithKline ; is indicated for the treatment of HIV-1 infection in combination with other antiretroviral agents.5 Resistance The most frequently observed viral mutations relating to abacavir have been K65R, L74V, Y155F, and M184V.5 In this nomenclature, the first letter represents the wild-type amino acid, the number indicates the amino acid position, and the subsequent letter corresponds to the amino acid of the mutant strain. In the K65R mutation, the mutant viral strain contains the amino acid arginine instead of lysine at position 65. Different mutations confer variable amounts of resistance to drug therapy, and multiple mutations may be necessary to confer significant resistance to drug therapy. Key primary mutations conferring resistance to ABC occur at positions 65, 74, 151, and 184. Multiple amino acid substitutions at positions 41, 67, 69, and 219 have also been associated with decreased susceptibility to ABC.7, 8 Didanosind ddI, Videx, Bristol-Myers Squibb ; , zalcitabine and dipyridamole.

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Table 4. Elements of Informed Consent for Prostate Cancer Screening. Chiong says: there is more emphasis now in nonpharmacologic and lifestyle modification, which wasn't really true more than 20 years ago and persantine. Name Age Date Please circle YES or NO to the following questions. Non-Modifiable Risk Factors: Yes No I have a family Mother, Aunt, or Sister ; history of broken bones or osteoporosis Yes No I have a small body frame and weigh less than 127 lbs. Yes No I have had a bone fracture as an adult after age 50. Yes No I female over the age of 65 Modifiable Risk Factors: Yes No I a smoker Yes No I currently do NOT take calcium supplements or eat foods rich with calcium Yes No I do NOT exercise for 30 minutes, 3 or more times a week If you have answered YES to 3 or more questions, you could be at risk for osteoporosis, and further evaluation may be needed. Speak to your health care provider to see if a bone density measurement may be warranted. Listed below are explanations as to why the above questions may put you at increased risk for osteoporosis. Susceptibility to fracture may be, in part, hereditary. Young women whose mothers have a history of vertebral fractures also seem to have reduced bone mass. A personal history of a fracture as an adult also increases fracture risk. Women with a small, thin frame and woman weighing less than 127 pounds are thought to be at greater risk due to less bone mass. The older one is the greater the risk of osteoporosis. Bones become weaker and less dense with age. The chance of developing osteoporosis is greater for females. Women have less bone tissue and lose bone more rapidly than men because of the hormonal changes involved in menopause. Current cigarette smoking, little or no weight-bearing exercise, and an inadequate intake of calcium increases the chance of developing osteoporosis. Smoking alters the body's absorption of calcium.Weight bearing exercise directly increases bone density. Produced by the Mid Atlantic Osteoporosis Board. For additional information visit or website at midatlanticosteo or the National Osteoporosis Foundation Website at NOF.

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Since the green white or blue white ; capsule of didanosine contains the drug as enteric-coated beadlets, it should be swallowed intact and not be opened, chewed or enshed and norpace. Felicio Rocho Hospital, Belo Horizonte, Minas Gerais, Brazil, and the University of Illinois Medical Center. Patients with sporadic tumors had no history of other endocrine tumors. The study was approved by the institutional review boards of the University of Illinois at Chicago, Felicio Rocho Hospital, and the other institutions, and informed consent was obtained from the subjects, for instance, didanosine and tenofovir!

ANTIRETROVIRALS NRTIs- abacavir Ziagen ; , abacavir lamivudine Epzicom ; , abacavir lamivudine zidovudine Trizivir ; , didanosine ddI, Videx , Videx EC ; , efavirenz emtricitabine tenofovir disproxil fumarate Atripla ; , emtricitabine Emtriva ; , lamivudine Epivir, 3TC ; , lamivudine zidovudine Combivir ; , stavudine d4T, Zerit ; , tenofovir Viread ; , tenofovir emtricitabine Truvada ; , zalcitabine ddC, Hivid ; , zidovudine AZT, Retrovir ; . PIs- amprenavir Agenerase ; , atazanavir Reyataz ; , darunavir Prezista ; , fosamprenavir Lexiva ; , indinavir Crixivan ; , lopinavir ritonavir Kaletra ; , nelfinavir Viracept ; , ritonavir Norvir ; , saquinavir Fortovase, Invirase ; . NNRTIsdelavirdine Rescriptor ; , efavirenz Sustiva ; , nevirapine Viramune ; , tipranavir Aptivus ; . Other- hydroxyurea Hydrea ; . Entry Inhibitors- enfuvirtide Fuzeon ; . OI DRUGS PHS "A1 OI"s- acyclovir Zovirax ; , amphotericin B Fungizone ; , azithromycin Zithromax ; , cidofovir Vistide ; , clarithromycin Biaxin ; , clindamycin Cleocin ; , famciclovir Famvir ; , fluconazole Diflucan ; , flucytosine 5FC, Ancobon ; , fomivirsen, foscarnet Foscavir ; , ganciclovir Cytovene ; , isoniazid generic ; , itraconazole Sporonox ; , leucovorin calcium Wellcovorin ; , pentamidine Nebupent, Pentam ; , prednisone oral generic ; , probenecid, pyrimethamine Daraprim ; , pyrazinamide generic ; , ribavirin generic ; * , rifabutin Mycobutin ; , rifampim generic ; , sulfadiazine oral generic ; , TMP SMX Bactrim, Septra ; , valganciclovir Valcyte ; , valacyclovir and motilium. Lactic acidosis and severe hepatomegaly with steatosis, including fatal cases, have been reported with the use of nucleoside analogues alone or in combination, including didanosine and other antiretrovirals. Videx EC is the brand name of an extendedrelease version of the anti-HIV drug ddI didanosine ; . Videx EC belongs to the group of antiretroviral drugs commonly called "nukes, " or nucleoside analogue reverse transcriptase inhibitors. Other examples of nukes include AZT, abacavir ABC, Ziagen ; , 3TC, d4T and ddC. These drugs fight HIV infection by interfering with the life cycle of the virus. At each stage of this cycle, proteins called "enzymes" help the virus make copies of itself replicate ; . Some drugs can slow down or stop inhibit ; the actions of these enzymes. When these enzymes can't perform effectively, the virus does not replicate as efficiently. This reduces HIV's ability to damage the immune system and doxepin. Acs american cancer society; dhea dehydroepiandrosterone; fda food and drug administration; gras generally recognized as safe; nci national cancer institute.

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Statistics Canada Web site Elite II, Pitt et al. 3 Heart and Stroke Foundation of Canada Web site 4 focusonbloodpressure ; MedicineNet 5 Heart and Stroke Foundation of Canada Web site 6 Heart and Stroke Foundation of Canada Web site 7 The Changing Face of Heart Disease and Stroke in Canada 2000, p. 61 8 The Changing Face of Heart Disease and Stroke in Canada 2000, p. 51 and sinequan and didanosine, for instance, abacavir. Observed included hypoactivity and cyanosis in both rodent species and severe vomiting in dogs. In rabbits, significant mortality was seen at doses of ciprofloxacin 2500 mg kg. Mortality was delayed in these animals, occurring 10-14 days after dosing. In mice, rats, rabbits and dogs, significant toxicity including tonic clonic convulsions was observed at intravenous doses of ciprofloxacin between 125 and 300 mg kg. DOSAGE AND ADMINISTRATION - ADULTS CIPRO Tablets and Oral Suspension should be administered orally to adults as described in the Dosage Guidelines table. The determination of dosage for any particular patient must take into consideration the severity and nature of the infection, the susceptibility of the causative organism, the integrity of the patient's host-defense mechanisms, and the status of renal function and hepatic function. The duration of treatment depends upon the severity of infection. The usual duration is 7 to days; however, for severe and complicated infections more prolonged therapy may be required. Ciprofloxacin should be administered at least 2 hours before or 6 hours after magnesium aluminum antacids, or sucralfate, Videx didanpsine ; chewable buffered tablets or pediatric powder for oral solution, other highly buffered drugs, or other products containing calcium, iron or zinc. ADULT DOSAGE GUIDELINES Infection Urinary Tract Severity Acute Uncomplicated Mild Moderate Severe Complicated Mild Moderate Dose 250 mg 250 mg 500 mg 500 mg 500 mg 750 mg 500 mg 500 mg 750 mg 500 mg 750 mg 500 mg 500 mg 500 mg 250 mg 500 mg Frequency Usual Durations q 12 h single dose q 12 h Days 7 to 14 Days 7 to 14 Days 28 Days 7 to 14 days 7 to 14 days 10 days 7 to 14 Days 7 to 14 Days 4 to 6 weeks 4 to 6 weeks 7 to 14 Days 5 to 7 Days 10 Days single dose 60 Days.

13. Yerly S, Rakik A, Kinloch-de-Loes S, et al. Prevalence de la transmission de virus resistantg a la zidovudine en Suisse. Schweizerische Medizinische Wochenschrift 126: 1845-1848; 1996. D'Aquila RT, Johnson VA, Welles SL, et al. Zidovudine resistance and HIV-1 disease progression during antiretroviral therapy. Annals of Internal Medicine 122: 401-408; 1995. Montaner JSG, Schechter MT, Rachlis A, et al. Sidanosine compared with continued zidovudine therapy for HIV-infected patients with 200 to 500 CD4 cells mm3: a double-blind, randomized, controlled trial. Annals of Internal Medicine 123: 561-571; 1995. Conway B, Montessori V, Rouleau D et al. Primary lamivudine resistance in acute HIV infection. International Workshop on HIV Drug Resistance, Treatment Strategies and Eradication, St. Petersburg, Florida, USA. Abstract 103; 1997 17. Connor EM, Sperling RS, Gelbert R, et al. Reduction of maternal-infant transmission of human immunodeficiency virus type 1 with zidovudine treatment: Pedriatic AIDS Clinical Trials Group Protocol 076 Study Group. New England Journal of Medicine 331: 1173-1180; 1994. Goudsmit JAAP, de Ronde A, Ho DD, et al. Human immunodeficiency virus fitness in vivo: calculations based on a single zidovudine resistance mutation at codon 215 of reverse transcriptase. Journal of Virology 70: 5662-5664; 1996. Larder B, Kemp S and Harrigan P. Potential mechanism for sustained antiretroviral efficacy of AZT-3TC combination therapy. Science 269: 696-699; 1995. Back N, Nijhuis M, Keulen W, et al. Reduced replication of 3TC-resistant HIV-1 variants in primary cells due to a processivity defect of the reverse transcriptase enzyme. EMBO Journal 15: 4040-4049; 1996. Imrie A, Beveridge A, Genn W, et al. Transmission of human immunodeficiency virus type 1 resistant to nevirapine and zidovudine. Journal of Infectious Disease 175: 1502-1506; 1997. Wei X, Ghosh S, Taylor M, et al. Viral dynamics in human immunodeficiency virus type 1 infection. Nature 373: 117-122; 1995. Ho D, Neumann A, Perelson A, et al. Rapid turnover of plasma virions and CD4 lymphocytes in HIV-1 infection. Nature 373: 123-126; 1995. Coombs R. Welles S, Hooper C, et al. Association of plasma human immunodeficiency virus type 1 RNA level with risk of clinical progression in patients with advanced infection. Journal of Infectious Disease 174: 704-712; 1996. Holodniy M, Katzenstein D, Israelski D, et al. Reduction in plasma human immunodeficiency virus ribonucleic acid after dideoxynucleoside therapy as determined by the polymerase chain reaction. Journal of Clinical Investigation 88: 1755-1759; 1991. Katzenstein D, Holodniy M, Israelski D, et al. Plasma viremia in and vibramycin. Morse GD, Fischl MA, Shelton MJ, Cox SR, Driver M, DeRemer M, et al. Single-dose pharmacokinetics of delavirdine mesylate and didanosune in patients with human immunodeficiency virus infection. Antimicrob Agents Chemother 1997; 41: 169-74. Barry M, Howe JL, Ormesher S, Back DJ, Breckenridge AM, Bergin C, et al. Pharmacokinetics of zidovudine and dideoxyinosine alone and in combination in patients with acquired immunodeficiency syndrome. Br J Clin Pharmacol 1994; 37: 421-6. Sahai J, Gallicano K, Seguin I, Garber G, Cameron DW. Interaction between zidovudine ZDV ; and didan0sine ddI ; [abstract 82]. 34th Interscience Conference on Antimicrobial Agents and Chemotherapy, New Orleans. October 4-7, 1994. LeLacheur SF, Simon GL. Exacerbation of dideoxycytidine-induced neuropathy with dideoxyinosine. Journal of the Acquired Immune Deficiency Syndrome 1991; 4: 538-9. Seifert RD, Stewart MB, Sramek JJ, Conrad J, Kaul S, Cutler NR. Pharmacokinetics of co-administered didanosine and stavudine in HIVseropositive male patients. Br J Clin Pharmacol 1994; 38: 405-10. Dicenzo R, Forrest A, Squires KE, Hammer SM, Fischl MA, Wu H, et al. Indinavir, efavirenz, and abacavir pharmacokinetics in human immunodeficiency virus-infected subjects. Antimicrob Agents Chemother 2003; 47: 1929-35. Dupont Pharma. Efavirenz Sustiva ; Product Monograph. Mississauga, ON: 2000 Ramanathan S, al. E. Lack of clinically relevant drug interactions between the ritonavir-boosted HIV integrase inhibitor GS-9137 elvitegravir ; and stavudine, didanosine or abacavir [abstract 70]. 8th International Workshop on Clinical Pharmacology of HIV Therapy, Budapest, Hungary. April 16-18, 2007. Wang LH, Blum MR, Hui J, Hulett L, Chittick GE, Rosseau F. Lack of significant pharmacokinetic interactions between emtricitabine and other nucleoside antivirals in healthy volunteers [abstract A 505]. 41st Interscience Conference on Antimicrobial Agents and Chemotherapy, Chicago, IL. December 16-19, 2001: 18. Hoechst Marion Roussel. Allegra Product Monograph. 2000 Astra Pharmaceutical Products Inc. Foscavir Product Monograph. Westborough, MA: 2000 Sahai J, Gallicano K, Pakuts A, Cameron DW. Effect of fluconazole on zidovudine pharmacokinetics in patients infected with human immunodeficiency virus [see comments]. J Infect Dis 1994; 169: 1103-7. Bruzzese VL, Gillum JG, Israel DS, Johnson GL, Kaplowitz LG, Polk RE. Effect of fluconazole on pharmacokinetics of 2', 3'-didioxyinosine in persons seropositive for human immunodeficiency virus. Antimicrob Agents Chemother 1995; 39: 1050-3. Damle B, Hess H, Kaul S, Knupp C. Absence of clinically relevant drug interactions following simultaneous administration of didanosineencapsulated, enteric-coated bead formulation with either itraconazole or fluconazole. Biopharm Drug Dispos 2002; 23: 59-66. Piscitelli SC, Kelly G, Walker RE, Kovacs J, Falloon J, Davey RT, Jr., et al. A multiple drug interaction study of stavudine with agents for opportunistic infections in human immunodeficiency virus-infected patients. Antimicrob Agents Chemother 1999; 43: 647-50. Hochster H, Dieterich D, Bozzette S, Reichman RC, Connor JD, Liebes L, et al. Toxicity of combined ganciclovir and zidovudine for cytomegalovirus disease associated with AIDS. Ann Intern Med 1990; 113: 111-7. Cimoch PJ, Lavelle J, Pollard R, Griffy KG, Wong R, LTarnowski T, et al. Pharmacokinetics of oral ganciclovir alone and in combination with zidovudine, didanosine, and probenecid in HIV-infected subjects. Journal of the Acquired Immune Deficiency Syndrome 1998; 17: 227-34. Jung D, Griffy K, Dorr A, Raschke R, Tarnowski TL, Hulse J, et al. Effect of high-dose oral ganciclovir on didanosine disposition in human immunodeficiency virus HIV ; -positive patients. J Clin Pharmacol 1998; 38: 1057-62. Frascino RJ, Gaines Griffy K, Jung D, Yu S. Multiple dose crossover study of IV ganciclovir induction dose 5 mg kg IV q12h ; and didanosine 200 mg po q12h ; in HIV-infected persons [abstract A-27]. 35th Interscience Conference on Antimicrobial Agents and Chemotherapy, San Francisco. September 17-20, 1995.
Absorption of ciprofloxacin is significantly reduced by concomitant administration of multivalent cation-containing products such as magnesium aluminum antacids, sucralfate, videx® didanosine ; chewable buffered tablets or pediatric powder, or products containing calcium, iron, or zinc see warnings : precautions, drug interactions and information for patients , and dosage and administration.

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This can help people to build long-term plans to maintain their health, for instance, protease inhibitors. Most people take abacavir without many side effects. Usually you will see a healthcare provider and have blood drawn in the first 2-4 weeks to look for the good effects of abacavir as well as any side effects. Possible side effects include mild nausea that usually gets better, ; headache, muscle aches, liver problems, or problems with blood cells. Remember most people will not have ANY side effects. Approximately 5% of patients who take abacavir develop allergy or "hypersensitivity" to it. This is a well-recognized and easy to treat situation. The allergic symptoms usually consist of SEVERAL of the following that always get worse as you take the drug: fever, body rash, cough, diarrhea, and nausea that gets worse. These symptoms may occur with other problems such as flu, food poisoning and allergies to other drugs. Therefore you should remember to inform your healthcare provider of any of these symptoms that tend to get worse and worse or more numerous as you continue to take abacavir. It is very important that you do not stop the abacavir unless instructed to by your healthcare provider or someone who is familiar with abacavir. If you have been diagnosed with hypersensitivity to abacavir, you must never take it again or you may die. All drugs of this class can cause or contribute to abnormal fat redistribution characterized by thinning of the face, arms, or legs. In most cases this would be also accompanied by elevated cholesterol levels, elevated triglyceride levels, and perhaps a tendency to develop diabetes. Abacavir does not seem to cause these problems very often. Rarely, a build-up of lactic ; acid may occur due to taking medications of this type. Persons taking multiple nukes NRTIs ; , those taking d4T stavudine, Zerit ; , those on the combination of d4T stavudine, Zerit ; and ddI didanosine, Videx ; , and those persons with hepatitis C are the most likely to encounter this rare, but potentially fatal problem. The symptoms are vague but troublesome: nausea, vomiting, muscle aches, weakness, turning yellow with jaundice, and just feeling plain bad and videx. TA-32. PHASE II TRIAL OF IODINE 131LABELED MURINE ANTITENASCIN MONOCLONAL ANTIBODY 81C6 M81C6 ; VIA SURGICALLY CREATED RESECTION CAVITIES IN THE TREATMENT OF PATIENTS WITH RECURRENT MALIGNANT BRAIN TUMORS D. Reardon, G. Akabani, A. Friedman, H. Friedman, J. Herndon, R. McLendon, J. Quinn, J. Rich, J. Vredenburgh, K. Penne, J. Sampson, T. Shafman, T. Wong, R. Coleman, M. Zalutsky, and D. Bigner; Duke University Medical Center, Durham, NC, USA Previously we established the maximum tolerated dose of iodine 131labeled murine antitenascin monoclonal antibody 81C6 131I-81C6 ; injected into the surgically created resection cavity of patients with recurrent brain tumors to be 100 mCi. In the current phase 2 study we have treated 40 patients with recurrent brain tumors, including 30 with glioblastoma multiforme GBM ; , five with anaplastic astrocytoma, two with anaplastic oligodendroglioma, 2 with progressive glioma, and one with metastatic adenocarcinoma. Patients were eligible if they underwent a gross total resection, had a Karnofsky performance score of 60%, and had adequate bone marrow, hepatic and renal function. The median age was 54.5 years range, 2573 ; . Twenty-eight 70% ; were male. All patients received prior external beam radiotherapy, and 14 35% ; had also received prior chemotherapy. All patients received a 100-mCi dose of 131I-m81C6 except for one patient who received 67 mCi due to a small resection cavity. Grade 3 or greater toxicity within 4 months of 131I-81C6 was of the following types: infection n 5 ; , hematologic n 4 ; , and neurologic n 4 ; . Delayed neurotoxicity 4 months after 131I-81C6 ; has occurred in only one patient to date. No patient has required reoperation to debulk radionecrosis. Median survival for all patients and those with recurrent GBM is 50.3 and 58.7 weeks, respectively. As of May 1, 2003, fifteen patients remain alive with a median follow-up of 25.1 weeks range, 5394 weeks ; . We conclude that 131I-m81C6 administered at a dose of 100 mCi is well tolerated and provides a clear survival advantage for patients following progression from conventional therapy. Montaner et al, a randomized, double-blind trial comparing combinations of nevirapine, didanosine, and zidovudine for hiv-infected patients for the incas study group, published jama; vol.

Please refer to the enclosed full prescribing information for Videx EC didanosine ; Delayed Release Capsules Enteric Coated Beadlets and Sustiva efavirenz ; Capsules and Tablets. BMS is committed to providing you with current product information for the management of your patients with HIV infection. If you have any questions about this new information or require additional medical information, please contact the Virology Medical Services Department at Bristol-Myers Squibb Company at 1-800-426-7644 select Option 3 ; . Sincerely, Sally L. Hodder, MD Vice President, Virology Medical Affairs, BMS!

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